ROUNDS·
Nephrology

AKI Approach: Pre-renal, Intrinsic, and Post-renal

How to classify acute kidney injury by cause, and which labs help you tell pre-renal apart from intrinsic damage.

KDIGO Definition

AKI is defined as a creatinine rise of 26.5 micromol/L (0.3 mg/dL) or more within 48 hours, a rise to 1.5 times baseline within 7 days, or urine output below 0.5 mL/kg/h for 6 hours.

The Three Categories

Labs That Help Differentiate

TestPre-renalIntrinsic (ATN)
FENa<1%>2%
Urine sodium<20 mEq/L>40 mEq/L
BUN:Creatinine ratio>20:1<15:1
UrinalysisNormal / hyaline castsMuddy brown casts, tubular cells

Initial Workup

Urinalysis, renal ultrasound (rule out obstruction first — it's reversible and easy to miss), a full medication review to identify and stop nephrotoxins, volume status assessment, and FENa where indicated.

Frequently Asked Questions

Why check for obstruction first?

Post-renal AKI from obstruction is often quickly reversible once relieved, and a renal ultrasound is quick, safe, and non-invasive — it's a high-yield, low-risk first step.

Is FENa reliable in every patient?

No — it's not valid if the patient has recently received diuretics, since diuretics independently increase sodium excretion. FEUrea is used instead in that situation.

What's the most common cause of intrinsic AKI?

Acute tubular necrosis (ATN), usually from prolonged ischemia (untreated pre-renal AKI) or nephrotoxic exposure (certain drugs, contrast, myoglobin).